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In addition to the canonical molecules in the Wnt/-catenin signalling pathway mentioned above, many other molecules also affect the signal transduction of the Wnt/-catenin signalling pathway
In general, three different groups of agents were described in the past: (i) mAbs, either targeting the ligand HGF or the receptor Met, (ii) tyrosine kinase inhibitors, targeting the tyrosine kinase domain of Met (and mostly also the TK domain of other RTKs), (iii) a truncated, soluble Met receptor serving as decoy for HGF (44) and the competitive HGF antagonist NK4 (18)
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Elevated ROS formation in the vessel wall is a key feature of cardiovascular disease and contributes to endothelial dysfunction, vascular inflammation, and cardiovascular remodeling (6)
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